Linggo, Hulyo 24, 2011

therapy treatment for stroke and bypass victim

jesoga70therapy medical mission/light of divine shadows 
0919606 0022 /226 27 24
www.jesoga70therapymedicalmission.webs.com

Coronary artery bypass surgery

how jesoga70therapy works to bypass/stroke patient
01. mr. juan d la cruz / bypass victim recovered by energizing the heart. by touching the 14 vitality abdominal impluses functioning system. residing at 300 sto rosario st., mandaluyong city . construction of blood circulation into the ventricles relax  heartbeat  cycles stay normal . in one single touch jesoga therapy proven speed healing done., the  therapy still going on the patient seem's batista brother (bodysized)

2) Ms juan D la Cruz 76 yrs of age from Zamboanga city , temporary residing at mb 27 rm112 suffer from heart problem and schedule 4 bypass operation at V. luna hospital at Quezon city that day when the family notify us because the operation was being cancelled due to hypertension. 9pm that night the family called us because the patient had shortness of breathings and they confirm it's to late to rush the patient to the hospitals. The situation really serious the patient is not talking anymore but the jesoga70therapy technique was trained mostly at these kind of retrival life saving condition. It's really a meracles  for the family after touching there 14 vitality abdominal impulses the patient went up and find the restroom (for bowel ) and speak to them , she's wandering how the touch's energize the heart muscle and stimulate the normal blood circulation take away the accumulation cholesterol in the blood stream, stop bleeding and promote the healing just a matter of 10 seconds. My dear view the family came from the marine, and phil nationa police forces ., these is the true story how jesoga70therapy is working to the human body(fatal) ? even the neighborhood exactly witness these testimony , we really want to secure every little pieces of our reliable information to the best of our ability to served the poor people from their badhealth and financially sufferings/loss. So anyone from you , is welcome to investigate these case whatever entention  theroff.(kuya neck) who own the unit. PnP personnel from camp crame Quezon city.
 From.
"Heart bypass" redirects here. For the technique to take over the function of the heart and lungs during surgery, see Cardiopulmonary bypass.
Coronary artery bypass surgery
Intervention

Early in a coronary artery bypass surgery during vein harvesting from the legs (left of image) and the establishment of bypass (placement of the aortic cannula) (bottom of image). The perfusionist and heart-lung machine (HLM) are on the upper right. The patient's head (not seen) is at the bottom.
ICD-10-PCS021209W
ICD-9-CM36.1
MeSHD001026

Coronary artery bypass surgery during mobilization (freeing) of the right coronary artery from its surrounding tissue, adipose tissue (yellow). The tube visible at the bottom is the aortic cannula (returns blood from the HLM). The tube above it (obscured by the surgeon on the right) is the venous cannula (receives blood from the body). The patient's heart is stopped and the aorta is cross-clamped. The patient's head (not seen) is at the bottom.

Three coronary artery bypass grafts, a LIMA to LAD and two saphenous vein grafts - one to the right coronary artery (RCA) system and one to the obtuse marginal (OM) system.
Coronary artery bypass surgery, also coronary artery bypass graft (CABG pronounced cabbage) surgery, and colloquially heart bypass or bypass surgery is a surgical procedure performed to relieve angina and reduce the risk of death from coronary artery disease. Arteries or veins from elsewhere in the patient's body are grafted to the coronary arteries to bypass atherosclerotic narrowings and improve the blood supply to the coronary circulation supplying the myocardium (heart muscle). This surgery is usually performed with the heart stopped, necessitating the usage of cardiopulmonary bypass; techniques are available to perform CABG on a beating heart, so-called "off-pump" surgery.
History
The first coronary artery bypass surgery was performed in the United States on May 2, 1960, at the Albert Einstein College of Medicine-Bronx Municipal Hospital Center by a team led by Dr. Robert Goetz and the thoracic surgeon, Dr. Michael Rohman with the assistance of Dr. Jordan Haller and Dr. Ronald Dee.[1][2] In this technique the vessels are held together with circumferential ligatures over an inserted metal ring. The internal mammary artery was used as the donor vessel and was anastomosed to the right coronary artery. The actual anastomosis with the Rosenbach ring took fifteen seconds and did not require Cardio-Pulmonary bypass. The disadvantage of using the internal mammary artery was that, at autopsy nine months later, the anastomosis was open, but an atheromatous plaque had occluded the origin of the internal mammary that was used for the bypass.[citation needed][verification needed]
Russian cardiac surgeon, Dr. Vasilii Kolesov, performed arguably the first successful internal mammary artery–coronary artery anastomosis in 1964.[3][4]
However, Goetz's has been cited by others, including Kolesov,[5] as the first successful human coronary artery bypass.[6][7][8][9][10][11][12] Goetz's case has frequently been overlooked. Confusion has persisted for over 40 years and seems to be due to the absence of a full report and to misunderstanding about the type of anastomosis that was created. The anastomosis was intima-to-intima, with the vessels held together with circumferential ligatures over a specially designed metal ring. Kolesov did the first successful coronary bypass using a standard suture technique in 1964, and over the next five years he performed 33 sutured and mechanically stapled anastomoses in St. Petersburg, Russia.[13][14]
Dr. René Favaloro, an Argentine surgeon, achieved a physiologic approach in the surgical management of coronary artery disease—the bypass grafting procedure—at the Cleveland Clinic in May 1967.[4][15] His new technique used a saphenous vein autograft to replace a stenotic segment of the right coronary artery. Later, he successfully used the saphenous vein as a bypassing channel, which has become the typical bypass graft technique we know today; in the U.S., this vessel is typically harvested endoscopically, using a technique known as endoscopic vessel harvesting (EVH). Soon Dr. Dudley Johnson extended the bypass to include left coronary arterial systems.[4] In 1968, Doctors Charles Bailey, Teruo Hirose and George Green used the internal mammary artery instead of the saphenous vein for the grafting.[4]

 Terminology

There are many variations on terminology, in which one or more of "artery", "bypass" or "graft" is left out. The most frequently used acronym for this type of surgery is CABG (pronounced 'cabbage'),[16] pluralized as CABGs (pronounced 'cabbages'). More recently[when?] the term aortocoronary bypass (ACB) has come into popular use. CAGS (Coronary Artery Graft Surgery, pronounced phonetically) should not be confused with Coronary Angiography (CAG).
Arteriosclerosis is a common arterial disorder characterized by thickening, loss of elasticity, and calcification of arterial walls, resulting in a decreased blood supply.
Atherosclerosis is a common arterial disorder characterized by yellowish plaques of cholesterol, lipids, and cellular debris in the inner layer of the walls of large and medium-sized arteries.

 Number of bypasses

The terms single bypass, double bypass, triple bypass, quadruple bypass and quintuple bypass refer to the number of coronary arteries bypassed in the procedure. In other words, a double bypass means two coronary arteries are bypassed (e.g. the left anterior descending (LAD) coronary artery and right coronary artery (RCA)); a triple bypass means three vessels are bypassed (e.g. LAD, RCA, left circumflex artery (LCX)); a quadruple bypass means four vessels are bypassed (e.g. LAD, RCA, LCX, first diagonal artery of the LAD) while quintuple means five. Bypass of more than four coronary arteries is uncommon.
A greater number of bypasses does not imply a person is "more sick", nor does a lesser number imply a person is "healthier."[17] A person with a large amount of coronary artery disease (CAD) may receive fewer bypass grafts owing to the lack of suitable "target" vessels. A coronary artery may be unsuitable for bypass grafting if it is small (< 1 mm or < 1.5 mm depending on surgeon preference), heavily calcified (meaning the artery does not have a section free of CAD) or intramyocardial (the coronary artery is located within the heart muscle rather than on the surface of the heart). Similarly, a person with a single stenosis ("narrowing") of the left main coronary artery requires only two bypasses (to the LAD and the LCX). However, a left main lesion places a person at the highest risk for death from a cardiac cause.[citation needed]
The surgeon reviews the coronary angiogram prior to surgery and identifies the lesions (or "blockages") in the coronary arteries. The surgeon will estimate the number of bypass grafts prior to surgery, but the final decision is made in the operating room upon examination of the heart.

 Indications for CABG

Several alternative treatments for coronary artery disease exist. They include:
Both PCI and CABG are more effective than medical management at relieving symptoms,[18] (e.g. angina, dyspnea, fatigue). CABG is superior to PCI for some patients with multivessel CAD[19][20]
The Surgery or Stent (SoS) trial was a randomized controlled trial that compared CABG to PCI with bare-metal stents. The SoS trial demonstrated CABG is superior to PCI in multivessel coronary disease.[19]
The SYNTAX trial was a randomized controlled trial of 1800 patients with multivessel coronary disease, comparing CABG versus PCI using drug-eluting stents (DES). The study found that rates of major adverse cardiac or cerebrovascular events at 12 months were significantly higher in the DES group (17.8% versus 12.4% for CABG; P=0.002).[20] This was primarily driven by higher need for repeat revascularization procedures in the PCI group with no difference in repeat infarctions or survival. Higher rates of strokes were seen in the CABG group.
The FREEDOM (Future Revascularization Evaluation in Patients With Diabetes Mellitus—Optimal Management of Multivessel Disease) trial will compare CABG and DES in patients with diabetes. The registries of the nonrandomized patients screened for these trials may provide as much robust data regarding revascularization outcomes as the randomized analysis.[21]
A study comparing the outcomes of all patients in New York state treated with CABG or percutaneous coronary intervention (PCI) demonstrated CABG was superior to PCI with DES in multivessel (more than one diseased artery) coronary artery disease (CAD). Patients treated with CABG had lower rates of death and of death or myocardial infarction than treatment with a coronary stent. Patients undergoing CABG also had lower rates of repeat revascularization.[22] The New York State registry included all patients undergoing revascularization for coronary artery disease, but was not a randomized trial, and so may have reflected other factors besides the method of coronary revascularization.
The 2004 ACC/AHA CABG guidelines state CABG is the preferred treatment for:[23]
The 2005 ACC/AHA guidelines further state: CABG is the likely the preferred treatment with other high-risk patients such as those with severe ventricular dysfunction (i.e. low ejection fraction), or diabetes mellitus.[23]

www.jesoga70therapymedicalmission.blogpost.com: therapy treatment for Andropause male or female me...

www.jesoga70therapymedicalmission.blogpost.com: therapy treatment for Andropause male or female me...: "jesoga70therapy and jin shin jyutsu therapy is the most elegant and unique powerfull knukles technique using the circular movement from ..."

www.jesoga70therapymedicalmission.blogpost.com: therapy treatment for stuttering victim for 10yrs ...

www.jesoga70therapymedicalmission.blogpost.com: therapy treatment for stuttering victim for 10yrs ...: "jesoga70therapy services 0919 606 0022 / 226 27 24 /0933 962 90 49 jesoga70therapy service mission therapy treatment for stuttering 09..."

Biyernes, Hulyo 15, 2011

therapy treatment for stuttering victim for 10yrs time og suffering





jesoga70therapy services
0919 606 0022 / 226 27 24 /0933 962 90 49
jesoga70therapy service mission
therapy treatment for stuttering
0919 606 00 22 / 226 27 24 /0910 372 19 76
www.jesoga70therapymedicalmission.webs.com
by Jesus Ogayre on Friday, July 1, 2011 at 7:36pm

stuttering (or stammering, as it is often called in Britain) is probably the best known and most researched speech disorder, but perhaps the most difficult to define, to explain, and to treat, especially in adults. Both names are onomatopoeic: the essential features of stuttering are frequent repetitions and prolongations of sound or syllables. Other problems of fluency may also characterize stuttering, including blocking of sounds or interjection of words or sounds. However, the sudden sensation of a loss of control over the ability to produce an utterance distinguishes stuttering from other forms of unfluent speech. Another characteristic of stutterers, especially adults, is their avoidance of anticipated problem words and situations, in order to cope with the problem.

Chronic stuttering usually begins in early childhood (development stuttering), although occasionally the disorder starts in adulthood (acquired stuttering), usually as a result of brain damage. Stuttering seems to occur in all nationalities with a prevalence of approximately 1% and an incidence of 4% to 5%. Approximately 40% to 80% of children with the disorder recover, for various reasons, by the time they reach adolescence or adulthood. More males than females stutter: the ratio of males to female stutterers ranges from 2-3: 1 in childhood, up to 4-5: 1 by adulthood. The incidence of the disorder is much higher among other members of the family of a stutterer than in the general population: in other words, there is probably a genetic factor in this condition.

Stuttering may vary greatly in its frequency and severity in different situations. It is, for instance, dramatically reduced when speaking alone or reading aloud in chorus with an accompanist. Wearing headphones that alter the feedback of one's own voice can also reduce stuttering, and so too can the use of certain regular speech patterns (e.g. singing, unusual accents, speaking in rhythm). The reliability with which such techniques for inducing fluency can reduce or even abolish stuttering is considered a key to understanding the disorder — they also form the basis of some treatments.

The social and vocational effects of chronic stuttering may be quite devastating, perhaps because of the suspicion that it is the outward sign of a personality disorder. However, there is remarkably little evidence that stuttering is related to any unusual personality characteristics or neuroses; and there is little support for the claim that stuttering is associated with anxiety. Many prominent individuals, including well-known actors and politicians, have managed to control their stuttering and achieve successful careers. Nevertheless, the handicapping effects of the disorder in children and adults are undeniable.

There is currently no accepted theory that offers a satisfactory explanation for all the features of stuttering. At different times, psychoanalytic or organic theories have held sway over research and/or therapy. Traditional, formal learning has been blamed; so too have errors in control systems in the brain. Most current researchers consider developmental stuttering to be a disorder of motor control, with strong genetic underpinnings, influenced by environmental factors. This position has gained support with evidence that signs of severe stuttering may appear almost as soon as a child starts to produce connected speech.

Recent studies of right-handed adult stutterers, using techniques for imaging activity in the brain, have revealed unusual patterns of activation and inhibition during stuttered speech, particularly in parts of the right hemisphere involved in hearing and the control of movement, and especially exaggerated in the cerebellum. The unusual activity in these regions, which appears to occur only during speech, is very different when stuttering is reduced or absent as a result of strategies for improving fluency. These findings have intensified the search for a neurological system or systems that might be specifically related to stuttering.

The treatment of stuttering is an area of controversy. There is no evidence that any drug treatment is effective in removing stuttering in children or adults. The most convincing effects have been reported for behavioural treatments, although there is much debate about the evaluation of such therapy. Other forms of treatment emphasize learning to adjust to the disorder rather than removing the problematic behaviour.

Probably the most dramatic change to therapy for stuttering in the last decade is the use of mild verbal corrections for each occasion of stuttering, and verbal praise for periods of fluency, which has beneficial effects in treating young children. Recovery commonly occurs without treatment in the first year after onset, but this becomes less likely without intervention if the disorder persists. Indeed, there is an urgent need to correct the widely-held belief that children will recover from stuttering if their problem is merely ignored. Currently, the preferred therapies for older stutterers involve a combination of behavioural techniques and methods for training speech-motor strategies. However, there is no convincing evidence that these therapies result in complete recovery.
 The most successful therapeutic approaches for adults and children involve three general features: first, a method that establishes reduced stuttering or stutter-free speech under relatively controlled conditions; then a procedure for transferring that improvement beyond the treatment setting; and finally, strategies for maintaining that improvement. The most favoured techniques for establishing control over stuttering fall into three categories: behavioural methods based on rewarding performance; teaching stutterers to prolong their speech; and mechanical aids. These techniques are also often used in conjunction with control of the rate of speaking. Once improvement has been produced in the controlled conditions, ‘transfer’ procedures are used, which systematically introduce increasingly demanding speaking situations. The most successful maintenance procedures require intermittent management over periods lasting up to two or three years. Given the variability of stuttering across situations, over time, and with relatively slight alterations to the manner of speech production, stuttering therapy evaluation presents considerable challenges, which are now occupying the attention of clinical researchers.
 The modern technique of Jesoga70 therapy cure Stuttering.
well , these is Jesoga70therapy it's your sensory and motor neuron doesn't working anymore why ? they should be activated and  touch correctly the most advance procedure on  how to get brain chemical structure charging in the balance resting potential condition , by expert therapist from mental martial practitioner(caution) so fatal ,  more than what knowledge physical therapy  do that can harmonize the (dtr )deep tendon reflex's so paralysis goes to function, but how ? terminal impulses to the brain must react to send electro chemical  substance that is responsible to the repair of damage muscle or nerves to the affected areas of atrophy. these conductor of electro chemical impulses well travel down length of the damage cell what we called the abundance of good Sudium and potassium. The balance effect which once jesoga70therapy ignited  the abdominal impulses functioning system including stuttering disease. cured that's mr Ryan Nabo of kangin sto. nino 1st street paranaque city 10yrs suffering from muscle atrophy due to sport injury.jesoga70therapy save him a live with more confident   now he came salute  the most natural and excellent  meaningful lifestyle and mission of every apostolic of mental martial art by jesoga70therapy. mabuhay po kay mr aikero zun.
jesoga70therapyaguado the evidence on herbs medicine
Advise for those who want to believed  for these mission and you'll faith was complete reasonable things if   god is true to your life then you're welcome to be our brother and sister as witness of his miraculous healing divine power  the everlasting  life flows down to each and every one of us, shining   to the name of our lord jesus christ. please save our continuing ministry service the jesoga70therapy healing mission especially to those financially loss , needs attention which found no cure to their doctor  pls come with us and save in the fire of sickness. One of our witness was mr byan nabo 0921 488 33 94 30yrs of legal age 10 yrs suffered from these disease(stuttering) hard to speak , unknown cure from the field of medicine but looks even the neurological doctor can't do their function and duties why because of very low quality of education they have . They can define what is  all about the subject but they cant reintegrate the materials and the process so god is exactly telling  us his unreachable wisdom  being the supreme being., even from the jesoga70aquado herbs medicine that allevate underlying anxiety and stuttering overexcitted , we really promotes these product  and the therapeutic use of those herbs we got , the flower therapy and essence therapy ? while taking these product you can  , keep the vocal chords open and relax and it helps control stuturing . Imitation is the reason why we exist to  found people safe for all existing product they are patronazing.  jesoga70therapy is a traditional medication healing mission and  we do advice our follower to make their own herbs medicine if they want , we need to educate them to their own faith and belief be your own doctor that's why we want donors and associate who want to give their own profession to the rest of your life to save other lives being philanthropist. that's the mantra of jesoga70therapy mission was created by god. the new choices in natural healing .









Miyerkules, Hulyo 13, 2011

3 yrs post stroke salvage patient from numbers of doctor ended no physical improvement





jesoga70therapy medical mission
www.jesoga70therapymedicalmission.webs.com
0919 606 00 22
3yrs stroke victim no changes for such medications leading for impairment function in older people. when some one is stoke the blood flow to the brain is impair or stops completely. so these patient sudden fell the numbness or weakness of face and arms or legs especially on side of the body , sudden confusion or trouble speaking or understanding , sudden trouble seeing in one or both eyes , sudden trouble walking , dizziness , or loss of balance or coordination and headache with no known cause these condition by ms Juan d la cruz make her suffer for 3 yrs long waiting for the miracle so by jesoga70therapy she found cure she's pretty good now. so much calls and interview we received from the concern family's asking how jesoga70therapy works to the human body ? jesoga70therapy medical mission pattern on how the interruption of blood supply to the due to the obstruction or rupture of a brain artery possibly thinner and cleanser for good circulation function, these treatment the victim recover from their sickness. whatever cause they have from embolism , thrombosis ,hypertension , congenitally from birth , weak arterial walls, or aneurysm , head injury and many more factor of stroke. leave it to us god is always watching these mission. pray for our soul and spirit for the enlightenment of our body. for those patient attending the everyday therapy guarantee  walk in 2 month or 3 months time/ and for those scheduled for 3 x a week session 6 months  same can walk..these stroke patient at her 4th therapy she is able to kick the legs and do the normal exercise of lower extremities.

Sabado, Hulyo 9, 2011

therapy treatment for pulmonary embolism

jesoga70therapy medical mission service
0919 606 00 22 / 226 27 24

Pulmonary embolism

From: maybo malbog 
 
Pulmonary embolism
Classification and external resources

Chest spiral CT scan with radiocontrast agent showing multiple filling defects both at the bifurcation and in the pulmonary arteries.
ICD-10I26.
ICD-9415.1
DiseasesDB10956
MedlinePlus000132
eMedicinemed/1958 emerg/490radio/582
MeSHD011655
Pulmonary embolism (PE) is a blockage of the main artery of the lung or one of its branches by a substance that has travelled from elsewhere in the body through the bloodstream (embolism). Usually this is due to embolism of a thrombus (blood clot) from the deep veins in the legs, a process termed venous thromboembolism. A small proportion is due to the embolization of air, fat, talc in drugs of intravenous drug abusers or amniotic fluid. The obstruction of the blood flow through the lungs and the resultant pressure on the right ventricle of the heart leads to the symptoms and signs of PE. The risk of PE is increased in various situations, such as cancer or prolonged bed rest.
Symptoms of pulmonary embolism include difficulty breathing, chest pain on inspiration, and palpitations. Clinical signs include low blood oxygen saturation and cyanosis, rapid breathing, and a rapid heart rate. Severe cases of PE can lead to collapse, abnormally low blood pressure, and sudden death.
Diagnosis is based on these clinical findings in combination with laboratory tests (such as the D-dimer test) and imaging studies, usually CT pulmonary angiography. Treatment is typically withanticoagulant medication, including heparin and warfarin. Severe cases may require thrombolysis with drugs such as tissue plasminogen activator (tPA) or may require surgical intervention viapulmonary thrombectomy.


Clinical manifestation:
symtoms depend on the size of the thrombus and the area of the pulmonary artery artery occlusion.
1, DYSPNEA , IS THE MOST COMMON SYMPTOM . 2 ,TACHYPNEA IS THE MOST FREQUENT SIGN . 3 , CHEST PAIN IS COMMON USUALLY SUDDEN IN ONSET  AND PLEURITIC IN NATURE , IT CAN BE SUBSTERNAL AND MAY MIMIC AGINA PECTORIS. 4 , fEVER , TACHYCARDIA  , COUGH , DIAPHORESIS , HEMOPTYSIS , SYNCOPE , SHOCK , AND SUDDEN DEATH MAY OCCUR. 5 , MULTIPLE SMALL  EMBOLI IN THE TERMINAL PULMONARY ARTERIOLES SIMULATE SYMPTOMS OF BRONCHOPNEUMONIA OR HEART FAILURE.
PREVENTION:
1 , AMBULATION OR LEG EXERCISE IN THE PATIENTS ON BED REST  2 , anticoagulation therapy before abdominothoracic surgery and every 8 to 12 hrs until discharge to from the hospital 3 , application of intermittent leg compression devices.
jesoga70therapy planning and goal:
The major goal include oxygenation , absence of additional thrombus, effective pharmacologic therapy minimal chest pain and anxiety , positive postoperative status , and effective home and community rate.
targe of preventing thombu fomation
1, encourage early ambulation and active and passive leg exercise 2 , instruct patient to move legs in a pumping exercise  3 , advise patient to avoid prolong sitting , immobility  and constrictive clothing ., 3 do not permit dangling of legs and feet in adependent position  4., instruct the patient to place feet on floor or chair and to avoid crossing legs. 5 , do not leave intravenous catheter in veins for prolonged periods. 6 , advise patient to continue wearing antiembolism stockings as long as directed. 7 , recommend that patient change position regularly when traveling , walk occasionally and do active exercises of legs and ankles , 8 , advise patient to drink plenty of liquids. 9. , teach patient to report dark tarry stools immediately is all about how to promote home and community base therapy care.
Allevating anxiety
encourage patient to express feelings and concerns and answer question concisely and accurately , explain therapy and discribe how to recognize untoward efect early.